| LitaMooxica | Date: Thursday, 04 Apr 2013, 3:06 PM | Message # 1 |
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No interactions were reported. i also checked mucinex d and mucinex dm too, just in case.
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To exert its desired effect, a drug generally must travel through the bloodstream to its site of action, where it produces some change in an organ or tissue. the drug's effects then diminish as it is processed (metabolized) by enzymes and eliminated from the body. alcohol behaves similarly, traveling through the bloodstream, acting upon the brain to cause intoxication, and finally being metabolized and eliminated, principally by the liver. the extent to which an administered dose of a drug reaches its site of action may be termed its availability. alcohol can influence the effectiveness of a drug by altering its availability. typical alcohol-drug interactions include the following (7): first, an acute dose of alcohol (a single drink or several drinks over several hours) may inhibit a drug's metabolism by competing with the drug for the same set of metabolizing enzymes. this interaction prolongs and enhances the drug's availability, potentially increasing the patient's risk of experiencing harmful side effects from the drug. second, in contrast, chronic (long-term) alcohol ingestion may activate drug-metabolizing enzymes, thus decreasing the drug's availability and diminishing its effects. after these enzymes have been activated, they remain so even in the absence of alcohol, affecting the metabolism of certain drugs for several weeks after cessation of drinking (8). thus, a recently abstinent chronic drinker may need higher doses of medications than those required by nondrinkers to achieve therapeutic levels of certain drugs. third, enzymes activated by chronic alcohol consumption transform some drugs into toxic chemicals that can damage the liver or other organs. fourth, alcohol can magnify the inhibitory effects of sedative and narcotic drugs at their sites of action in the brain. to add to the complexity of these interactions, some drugs affect the metabolism of alcohol, thus altering its potential for intoxication and the adverse effects associated with alcohol consumption (7).
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